Neuroinflammation, a typical condition of AD brains, is, among others, sustained by MAO B, whose upregulation in reactive astrocytes results in aberrant GABA levels [8] and reactive oxygen species (ROS) production through the catalytic deamination of the starting amine substrate into an aldehyde metabolite and hydrogen peroxide as the byproducts [9]. This evidence concerns the gene MAOB and Alzheimer disease.