Comprehensive studies examining Cav-2 expression spectrum among different tumor-infiltrating bone marrow-derived cell types including (but not limited to) M1-like vs. M2-like TAMs, monocytes, polymorphonuclear leukocytes, NKT and NK cells during earlier vs. later stages of tumor growth will undoubtedly shed more light into better understanding of the mechanisms via which Cav-2 regulates tumor growth and anti-tumor immune response. Here, CAV2 is linked to neoplasm.