A granulocyte signature that includes inflammatory cytokines and DNA-binding antimicrobial peptides (LL-37 in human subjects and cathelicidin-related antimicrobial peptide (CRAMP) in mouse) is associated with aberrant neutrophil function, and the formation of NETs is increased in various autoimmune diseases including psoriasis [17, 19, 20, 43]. The gene discussed is CAMP; the disease is autoimmune disease.