Intriguingly, either overexpression of E2F1 or Myc could efficiently rescue the inhibition of cervical cancer cells’ proliferation or migration which induced by FTO’s deficiency, suggesting these two genes might regulate each other in cervical cancer cells, for instance, Myc was proved to induce transcription of E2F1 while inhibiting its translation via a microRNA polycistron [35]. Here, MYC is linked to cervical carcinoma.