The virus has been shown to utilize the receptor tyrosine kinase AXL and its ligand Gas6 for viral entry (Meertens et al., 2017), albeit contradictory findings on the role of the receptor in ZIKV infection were reported (Wells et al., 2016; Hastings et al., 2017; Li et al., 2017; Wang et al., 2017). This evidence concerns the gene NTRK1 and Zika virus infectious disease.