Danikowski et al. hypothesized that enhanced bone resorption and myogenic proliferation inhibition in MG patients might be related to dysfunction of T-regulatory cells (Tregs) and overexpression of cytokines and chemokines, such as tumor necrosis factor-α (TNF-α), interleukin-6 (IL-6), and 10 (IL-10) (32, 33). The gene discussed is IL6; the disease is myasthenia gravis.