Statins are capable of causing G0/G1 or G2/M arrest or activating intrinsic apoptotic pathway via regulating Bcl‐2 protein family.20, 31 Statins may also target histone deacetylases (HDACs) to alter protein acetylation status to reduce cell proliferation and in vivo tumour growth.20, 32 Characterizing statins’ anti‐tumour mechanisms will aid in their proper application as anti‐cancer therapeutics in the future. This evidence concerns the gene BCL2 and neoplasm.