The therapeutic landscape of non-small-cell lung cancer (NSCLC) harboring oncogenic driver alterations has revolutionized by the introduction of tyrosine kinase targeted inhibitors (TKIs), such as Epidermal Growth Factor Receptor (EGFR) [1], and Anaplastic Lymphoma Kinase (ALK) TKIs [2]. Here, ALK is linked to non-small cell lung carcinoma.