In line with our data showing that JAK2 phosphorylation was lower in HPV− cervical cancer cells, inhibition of JAK2 in HPV− C33A cells resulted in a dose-dependent reduction in JAK2 and STAT3 phosphorylation but had minimal effect on cell growth (Supplementary Figure S2A), anchorage-dependent (Supplementary Figure S2B) and anchorage-independent colony formation (Supplementary Figure S2C). Here, STAT3 is linked to cervical carcinoma.