In hypoxic region, basal autophagy was upregulated to facilitate survival of tumor.29 Meanwhile, autophagy‐regulated glioma‐initiating cells' self‐renewal and suppressed tumorigenicity depended on Notch1 degradation.30 Thus, our further investigation should focus on whether NEAT1 could regulate other progress in cancers via modulating autophagy. This evidence concerns the gene NOTCH1 and cancer.