In hypoxic region, basal autophagy was upregulated to facilitate survival of tumor.29 Meanwhile, autophagy‐regulated glioma‐initiating cells' self‐renewal and suppressed tumorigenicity depended on Notch1 degradation.30 Thus, our further investigation should focus on whether NEAT1 could regulate other progress in cancers via modulating autophagy. Here, NEAT1 is linked to central nervous system cancer.