MiR‐34a, one of the first identified tumor suppressor genes, commonly shows low expression in many tumors, such as breast cancer, lung cancer, and acute myeloid leukemia.31 MiR‐34a was involved in many cellular progress, like p53‐induced cell cycle arrest, apoptosis, and negatively regulated SIRT1.32 In the present study, NEAT1 acted as ceRNA of miR‐34a and further resulted in the elevation of downstream targets. This evidence concerns the gene NEAT1 and lung cancer.