This suggested that the innate immune response in ovarian tumors may be antagonized by ADAR1.33 To broaden the concept of immune suppression and establish a potential basis for the association of RIG‐I with unfavorable OC outcome, we determined the expression levels of checkpoint molecules34 as well as a fate‐specifying transcription factor of regulatory T cells (Tregs).35 We found that tumors with high RIG‐I expression were markedly enriched in PD‐L1 (p < 0.001, Fig. 4g) and PD‐1 mRNA expression (p = 0.002, Fig. 4h). Here, RIGI is linked to ovarian neoplasm.