Specifically angiopoietin-2, IL-8, and G-CSF, along with others reported in Tables 6–8 according to their cell-specific patterns with BPD, have been shown to vary in human models of neutropenic sepsis and death and may serve as predictors of response to therapy (41, 42). The gene discussed is CXCL8; the disease is bronchopulmonary dysplasia.