Despite conflicting microarray and RNA-sequencing results, previous work has repeatedly found significantly reduced NDUFS7 protein levels within the PFC of patients with BD, as well as reduced ETC complex I activity.10,17 These previous findings alongside our results lead us to believe that complex I subunit NDUFS7 and the PFC play a fundamental role within the pathology of BD and should be continuously studied in order to aid the discovery of the underlying mechanisms and improve treatment regimens for patients with BD. Here, NDUFS7 is linked to Behcet disease.