Diabetes is considered an important health‐related complication in humans that results when the pancreas cannot produce a sufficient amount of insulin (Type 1 diabetes) or when the body cannot effectively utilize the insulin which it makes (Type 2 diabetes) (T2D).1 It is estimated that by 2030, around 552 million people around the world will be diagnosed with T2D indicating the growing gravity of this health problem.2 Despite such disease burden associated with socioeconomic issues, the pathogenesis of T2D is not yet completely understood. This evidence concerns the gene INS and type 2 diabetes mellitus.