We found that while there was no increase in PD‐L1 expression by the monocytic myeloid‐derived suppressor cells (Mo‐MDSC; defined as live singlets with surface markers CD45+CD11b+CD11c−Ly6ChiLy6G−) subgroup, the neutrophilic myeloid‐derived suppressor cells (PMN‐MDSC; live singlets with surface markers CD45+CD11b+CD11c−Ly6CloLy6G+; Bronte et al., 2016) population displayed a significant elevation in PD‐L1 in the lungs of mice treated with both diphtheria toxin and bleomycin, compared to control animals (Figure 6c,d). This evidence concerns the gene ITGAM and diphtheria.