Notably, 87% of DEGs significantly upregulated in the diabetes-VS-normal group comparison were enriched in inflammation and immune-related processes, including inflammatory and immune response, the cytokine and chemokine pathway, JAK-STAT/nuclear factor κB (NF-κB)/NOD-like/Toll-like receptor signaling pathways, and apoptosis pathways (Figure 2C). The gene discussed is NFKB1; the disease is diabetes mellitus.