Compared the Data From Akt-CA With Those From ERK-CA Groups, ERK Activation Exerted Stronger Effect on Eliminating Palbociclib-Induced Inhibitive Effects, Suggesting a Major Role of ERK Signaling in Palbociclib-Induced Blockade of GBM (Figure 5B). The gene discussed is AKT1; the disease is glioblastoma.