Consistent with the data above, and in line with contemporary knowledge implicating increased glutathione (GSH) levels in tumor initiation, disease progression, increased metastasis, and the chemoresistant stem cell-like phenotype of cancerous cells [24,25,26], we showed that the median intracellular GSH level in the FAT1-rich HSC-3 CispR cells was 2.38-fold (p < 0.01) fold higher than in the HSC-3 wild-type cells (Figure 6D). This evidence concerns the gene FAT1 and neoplasm.