Individuals with pathogenic RAD51C variants in BC were previously reported to be primarily ductal, hormone receptor‐positive and HER2‐negative, or triple negative invasive carcinomas of no special type, diagnosed at an early stage with moderate differentiation (Gevensleben et al., 2014; Meindl et al., 2010; Schnurbein et al., 2013), and OC is primarily HGSC (Cunningham et al., 2014; Gevensleben et al., 2014). Here, ERBB2 is linked to invasive carcinoma.