Consistently, as shown in Figure 1(f), Oncomine databases demonstrated that the CKS2 expression gradually increased from normal livers (fold change, 4.12; P < 0.001), cirrhotic livers (fold change, 2.50; P < 0.001), and dysplasia livers (fold change, 1.94; P < 0.001) to HCC. This evidence concerns the gene CKS2 and dysplasia.