IL17A and Ureteral obstruction: In two models of experimental renal damage, unilateral ureteral obstruction and immune mediated glomerulonephritis by nephrotoxic serum, we described that BET inhibition by JQ1 treatment markedly diminished the presence of IL17A expressing cells in the injured kidneys, associated with downregulation of renal levels of IL17A and other Th17-related cytokines, such as CCL-20 and CSF-1 (Suarez-Alvarez et al., 2017).