In the complex network regulated by plants in response to external stress, there is a significant antagonistic relationship between SA and JA signalling pathways, studies have shown that SA and JA may inhibit each other′s synthesis and their downstream signalling pathways [37], moreover, node genes in SA and JA signalling pathways, such as NPR1 and SSI1, may play fine regulatory roles in their antagonistic effects [39], which confirms the completely different changes in SA and JA after infection with A. tenuissima in this experiment. The gene discussed is SOCS1; the disease is infection.