Constitutive Stat3 activation has intrinsic consequences on the tumour cell as well as effects within the extracellular matrix (ECM) and stromal cells of the tumour microenvironment, thus resulting in increased tumour cell proliferation, motility, survival and invasiveness, as well as tumour‐promoting activities of angiogenesis and evasion of tumour‐suppressing immunity.28 Based on its common activation in ovarian carcinoma and its extensive cellular activities, Stat3 has become an attractive therapeutic target. This evidence concerns the gene STAT3 and neoplasm.