However, while the peak of expression of IFN‐γ was observed at 2 d.p.i., the expression of IL‐17A continued increasing until 3 d.p.i. Furthermore, we analyzed by intracellular staining the expression of both cytokines in the two main γδ T cell subsets and we observed that both subsets significantly expressed IL‐17A and almost no IFN‐γ after infection (Fig. 4C, upper panels). The gene discussed is IL17A; the disease is infection.