Accordingly, we showed that the treatment with antibodies that generally deplete γδ T cells or antibodies that only deplete Vγ4+ γδ T cells reduced IL‐17A levels, which correlated with lower levels of CXCL1 and neutrophil recruitment at 3 d.p.i. Moreover, neutrophils are known to play a protective role during influenza infection by limiting influenza virus replication during the early and later phases of infection 41. This evidence concerns the gene CXCL1 and infection.