GATA3 and ulcerative colitis: Li et al. reported an increase in Lin+ GATA3+ as well as Lin− GATA3+ ILC2‐like populations together with a more pronounced type 2 signature limiting the protective type 1 immune response in the development of cancers.116 In mice, Helicobacter typhlonius infection triggers IL‐17A expressing ILCs by synergizing DCs derived TNF‐α and IL‐23 in the Tbx21−/−Rag2−/− ulcerative colitis (TRUC) model.83