The promotion effects of tumour growth and metastasis driven by SNHG15 overexpression can be significantly reversed by miR‐141 inhibitor.19 Besides, wnt/β‐catenin signalling pathway was found to be involved in SNHG15‐mediated carcinogenesis and could act as a downriver regulator in CRC.19 These findings suggest that SNHG15/miR‐141/SIRT1 axis exerts oncogenic functions in CRC.19 Similarly, Min et al found that SNHG15 mediated CRC proliferation through SNHG15/miR‐338‐3p/FOS/RAB14 axis.19 The gene discussed is FOS; the disease is colorectal carcinoma.