Further research confirmed that miR‐211‐3p can bind to SNHG15 and its downregulation can partly rescued the proliferation promotion driven by SNHG15 overexpression in A549 and H1799 cells.78 Moreover, SNHG15 could upregulate CDK14 expression via sponging miR‐486, thus contributing to NSCLC tumorigenesis.44 These findings elucidated that SNHG15 can activate the malignant phenotypes of NSCLC cells through a mechanism involving miRNAs. The gene discussed is SNHG15; the disease is non-small cell lung carcinoma.