Identical to a previous report, Bcl-2 immunostaining is cytoplasmic and granular and restricted in normal bronchial epithelium to the basal epithelial layer or to some epithelial cells that are perpendicularly oriented to the basal lamina before infection [53], whereas, after infection, the Bcl-2 protein expression in the bronchial epithelial cells almost vanished and only a few Bcl-2 were noted in the neutrophil-infiltrated areas. This evidence concerns the gene BCL2 and infection.