ZEB1 and infection: The infection of gastric epithelial cell lines by cagA-positive H. pylori strain induces morphological modifications characterized by a loss of polygonal shape, cell cluster disruption, elongated shape, stimulation of mesenchymal (Snail, vimentin, and Zeb1) and stem cell (CD44) markers, and inhibition of epithelial markers (cytokeratin 7 and osteopontin) [81].