This association is plausible, as (i) Universities in North America represent an obesity-prone environment for young adults22; (ii) SNPs in FTO are strongly associated with polygenic obesity in children and adults of European descent23; (iii) SNPs in FTO interact with obesity-prone environments to promote obesity24; (iv) an association between weight gain and FTO rs9939609 SNP (in strong linkage disequilibrium with rs3751812 in populations of European ancestry) has been previously reported in university students from the United Kingdom25. The gene discussed is FTO; the disease is Obesity.