It frequently occurs at the site of wall motion abnormalities of the apex.[32] The incidence of LVT was reported as 32% in patients with a LVEF of ≤30% and 3% in those with a LVEF of 50% to 59%.[33] The possible involvement of the factor V Leiden mutation in the onset of LVT after AMI was examined, but its involvement was not confirmed.[34] A case of essential thrombocythemia leading to AMI and subsequent LVT was also reported.[35] In the present case, LVT occurred despite preserved LVEF, suggesting the involvement of secondary thrombocytosis after cesarean section as an etiological factor. The gene discussed is F5; the disease is thrombocytosis disease.