HDAC9 and hepatocellular carcinoma: For example, p65 directly bound to the promoter region of miR‐590 and repressed its expression in osteosarcoma, a process independent of p65 phosphorylation.24 Alternatively, p65 might act like a scaffold protein and recruit HDAC to miR‐23a promoter, which in turn inhibited the expression of miR‐23a in leukemic Jurkat cells.25 We concluded that phosphorylation was not indispensable to p65‐mediated inhibition of miR‐23a expression in HCC.