Chen et al. unraveled the tumor-suppressive role of miR-199a-5p in TNBC associated with proliferation inhibition, cell cycle arrest, and increasing apoptosis by regulating the downstream targets of TGF-β2 and PIK3CD [83], and the overexpression of a novel tumor suppressor of miR-1296 involved in suppressed cell proliferation, cell cycle arrest, accompanied by induction of apoptosis by targeting cyclin D1 (CCND1) in TNBC cells [178]. The gene discussed is CCND1; the disease is neoplasm.