Furthermore, chronic, administration of BSO in combination with an inhibitor of DA transporter (DAT), the compound GBR 12909, during the early postnatal development (p5–p16) to Osteogenic Disorder Shionogi (ODS) mutant rats, induced the long-term cognitive deficits, assessed in the novel object recognition test (NOR) in adulthood [42,43]. The gene discussed is SLC6A3; the disease is Cognitive impairment.