An autoinflammatory disease response has been recently mentioned as a typical hallmark of neurodegenerative diseases like DM [71] and aberrant myokine and cytokine signaling activities, processes that are tightly coupled to IFNγ function, have been reported before, for both cDM muscle tissue and endocrine levels in the circulation of DM patients [72]. The gene discussed is IFNG; the disease is glycogen storage disease VI.