Another study by Wang et al indicated that BMSC-derived exosomes can promote the proliferation, migration, and survival of MM cells, induce drug resistance to bortezomib, and influence several signaling pathways including c-Jun N-terminal kinase, TP38, TP53, and AKT, which are relevant to the survival of MM cells 9. The gene discussed is AKT1; the disease is Miyoshi myopathy.