Utrophin is an autosomal paralogue of dystrophin and highly similar in sequence, size and function.12 Increasing utrophin in the dystrophin‐deficient mdx mouse model13 prevents the dystrophic phenotype,14 not only offering a potential therapy to all DMD patients regardless of mutation type but also avoiding an immune response.11, 15. This evidence concerns the gene UTRN and Duchenne muscular dystrophy.