Hypertriglyceridaemia in CKD is thought to be due to the dysregulation of various enzymes such as lipoprotein lipase (LPL) and hepatic lipase, apolipoproteins such as apoC III and apoC II and receptor such as VLDL-receptor involved in triglycerides metabolism [86]. This evidence concerns the gene APOC2 and hypertriglyceridemia.