We evaluated the inhibitory effect of NQTrp and Cl-NQTrp toward aggregation of Calcitonin, Insulin and Lysozyme aggregation (Scherzer-Attali et al., 2012b), which form amyloids in vivo and are implicated in medullar carcinoma of the thyroid, insulin injection amyloidosis and hereditary systemic amyloidosis, respectively (Pepys et al., 1993; Westermark et al., 2005; Guo et al., 2015). This evidence concerns the gene INS and amyloidosis.