AKT1 and coronary artery disorder: We demonstrated that ox‐LDL at the concentrations that were compatible with serum ox‐LDL levels or less than that in patients with stable coronary artery diseases 12, 45, 46, 47 induced significant membrane damage and substantially impaired the survival of MAPCs, and selectively decreased Akt phosphorylation without change in the levels of total Akt or total and phosphorylated STAT3 in MAPCs.