Since contribution of HSPC to other autoimmune blistering dermatoses including epidermolysis bullosa acquisita and possibly dermatitis herpetiformis was also found, more universal roles of HSPC as a pathophysiological factor and potential target for treatment of autoimmune BP diseases have been suggested (Tukaj et al. 2015). This evidence concerns the gene PSMA7 and dermatitis herpetiformis.