It has been suggested that miR‐130 expression interacting with Hox genes could control vascular morphogenesis in developing lung.32 The role of miR‐130a was characterised in reducing HOXA5 expression, thus decreasing p53 expression and controlling breast cancer cells resulting in tumour progression and metastasis.33 MiR‐130a attenuated endocrine disorders, ovarian injury and apoptosis of granulosa cells in rat models of PCOS. The gene discussed is TP53; the disease is polycystic ovary syndrome.