Zheng et al.117 revealed that lung cancer cells treated with X-ray radiation could release miR-23a with increased expression in EVs, mainly including exosomes, and the EV miR-23a taken up by HUVECs could promote the proliferation and migration of these recipient cells by restraining the expression of phosphatase and tensin homolog (PTEN), thereby enhancing angiogenesis and resistance to radiotherapy. Here, PTEN is linked to lung carcinoma.