AK2 and reticular dysgenesis: Taken together, these data indicate that, as previously observed for hematopoietic phenotypes, the administration of antioxidant treatment with GSH can partially reduce the sensorineural defects observed in an ak2-deficient zebrafish model of RD; confirming that similar treatment strategies could potentially be useful in ameliorating non-hematopoietic defects in RD patients.