These results might be on account of that lncRNA MVIH regulated some genes such as jun‐B and miR‐199a, which have been implicated as crucial regulators in the pathology of AML, and eventually enhanced AML cell proliferation but inhibit apoptosis.16, 17 Most importantly, our data implied that lncRNA MVIH might act as a possible treatment target in AML. Here, JUNB is linked to acute myeloid leukemia.