We included seven (pAKThigh) GCB-DLBCL cell lines with strong (BJAB, SU-DHL-5, OCI-Ly1, WSU-DLBCL2, Karpas-422, HT, DoHH2) and three with weak (WSU-NHL, OCI-Ly19, SU-DHL-4) (pAKTlow) AKT activity and compared the PI3K-PDPK1-dependent AKTT308 and auxiliary mTORC2-dependent AKTS473 phosphorylation signals [27, 28] with that of six BL cell lines. This evidence concerns the gene PDPK1 and diffuse large B-cell lymphoma.