A moderate increase in ROS levels stimulates the MAPK/Erk1/2, PI3K/Akt, and IKK/NF-κB pathways [6,7,8,9], which accelerates cell-cycle progression by upregulating the mRNA levels of cyclins [10], actively promotes cell survival through protein kinase D1 (PKD1) and Akt kinase [11], and positively affects many other factors, such as energy metabolism, cell morphology, cell–cell adhesion, cell motility, angiogenesis, and tumor stemness. This evidence concerns the gene NFKB1 and neoplasm.