It was then reported that autophagy genes from the ATG family, such as beclin1 and atg5, are overexpressed in CD34+ cells from chronic-phase CML patients [139] and could be repressed by miR-30a, whose expression decreased after Imatinib treatment [15]. The gene discussed is BECN1; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.