Typically, Campylobacter jejuni bearing the gangliosides‐like lipo‐oligosaccharides (LOS) accounts for the pathogenesis of axonal GBS, particularly acute motor axonal neuropathy.4 Cytomegalovirus (CMV) infection is associated with severe motor sensory deficits, demyelination, and antibodies to the ganglioside GM2.3Mycoplasma pneumoniae infection is associated with anti‐galactocerebroside (GalC) antibodies and pediatric GBS.5 Global variation in infection burden may at least in part explain the regional differences in clinical presentation and subtype of GBS. This evidence concerns the gene GALC and infection.