Utilization of glutamine for anabolic synthesis and the expression of genes associated with glutamine metabolism are upregulated in NIH3T3 cells expressing KRAS‐mutated proteins and in KRAS‐mutated human breast cancers.15 In pancreatic cancers, oncogenic KRAS plays an important role in the reprogramming of glutamine metabolism.16 Furthermore, glutamine catabolism in the TCA cycle is necessary for KRAS‐induced anchorage‐independent growth.17 Taken together, glutamine plays an essential role in the growth of KRAS‐mutated cells. The gene discussed is KRAS; the disease is pancreatic neoplasm.