Liu et al. [15] proposed that the oncogenic properties of miR-182 in ovarian cancer may be partly due to its impaired repair of DNA double-strand breaks, negative regulation of breast cancer 1 (BRCA1) and metastasis suppressor 1 (MTSS1) expressions, and positive regulation of oncogene high-mobility group AT-hook 2 (HMGA2). This evidence concerns the gene BRCA1 and ovarian carcinoma.