AKT1 and cervical cancer: For example, one study has shown that over-expression of miR-182 inhibits the epithelial to mesenchymal transition and metastasis via inactivation of Met/AKT/Snail in non-small cell lung cancer cells [13], Sun et al. [14] have reported that the miR-182 expression in cervical cancer is down-regulated and miR-182 induces cervical cancer cell apoptosis by suppressing DNMT3a expression.